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GLP-1 binding to GLP-1R promotes -cell glucose metabolism through mTOR-dependent HIF-1 activation

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Furthermore, GLP-1 exhibits significant cardioprotective properties, particularly in pathological conditions, such as ischemia and myocardial damage

First, it is likely that some compounds are administered in preclinical studies at doses that may be much higher on a relative basis than those tolerated in human studies

Glucagon-like peptide-1 increases myocardial glucose uptake via p38alpha MAP kinase-mediated, nitric oxide-dependent mechanisms in conscious dogs with dilated cardiomyopathy

Today, approximately 50% of all HF patients demonstrate preserved ejection fraction, and the prevalence of this condition continues to increases annually