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Since podocyte injury extends beyond Gb3 accumulation, analysis of signaling networks in Fabry have shown the involvement of -synuclein in lysosomal impairment and hints to pharmacological treatments modulating -synuclein and the use of 2 -adrenoreceptor agonists [65] as promising interventions

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Proc Natl Acad Sci U S A 89: 8641-8645, 1992