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Consistent with this idea, protein carbonylation, considered the hallmark for oxidative protein damage and detected using 1,3-dinitrophenol (DNP) flow cytometry, was not increased in G6PDH-null chondrocytes, indicating no manifest increase in oxidative protein damage (Fig
Z and S mutations of the 1-antitrypsin gene and the risk of chronic obstructive pulmonary disease
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Role of thioredoxin reductase 1 and thioredoxin interacting protein in prognosis of breast cancer
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