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The big reason why is that the market was widely expecting approval to happen, given how well the drug was performing in clinical trials

doi: 10.1155/2006/248387

Oleoylethanolamide (OEA), an ethanolamide fatty acid synthesized in the body, can regulate glucose homeostasis and weight loss by stimulating GLP-1 secretion from intestinal L-cells and insulin secretion from -cells in a GPR119-dependent manner and by affecting the abundance of the bacterium Akkermansia muciniphila [146, 147]

Furthermore, activation of CB2 is devoid of any psychotropic effects [62]

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