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showed that the activation of ferroptosis by erastin increased the levels of lysosome-associated membrane protein 2a (LAMP2A) to promote CMA, which, in turn, triggered the degradation of GPX4

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discovered that the antioxidant enzyme GPX-1 might be a possible therapeutic target for treating cigarette smoke-induced lung disease because of the increased oxidant burden in smokers and patients with COPD [30]

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