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Infections may also contribute to HT via molecular mimicry, in which structural or amino acid sequence similarities between microbial and human antigens induce cross-reactive immune responses, accelerating the development of autoimmunity (da Silva et al

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Finally, we highlight major limitations in the current evidencesuch as heterogeneous dosing regimens, the scarcity of long-term clinical data, and insufficient mechanistic validation under MASLD conditionsand propose directions for future translational research

Nephrology , 2 , 17583