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Furthermore, for the first time, we revealed that PQQ could ameliorate the memory deficits and neurotoxicity induced by D-gal via binding with excess glutamate, inhibiting oxidative stress, and eliminating inflammation through GSK-3/Akt signaling pathway

Here, we revealed significant decrease of Haem oxygenase (HMOX1) and Serotransferrin (TF) and significant enrichment of Glutathione synthetase (GSS) and ceruloplasmin (CP) in AD-NPCs compared to controls (Supplementary Fig

Examples include the synthesis of nucleotides and non-essential amino acids

doi: 10.1371/journal.pone.0082105

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