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10.1021/ja100726a J

RA and depression exhibit shared lipid-mediated inflammatory mechanisms driven by dysregulated pro-inflammatory and impaired pro-resolving pathways (200, 302)

NAD+ Preservation and Sirtuin Activation First and most directly, NNMT activity consumes nicotinamide, which is a key precursor for NAD+ biosynthesis through the salvage pathway (the predominant NAD+ synthesis route in most mammalian cells)

Preso regulates NMDA receptor-mediated excitotoxicity via modulating nitric oxide and calcium responses after traumatic brain injury

Crucially, the specific papers tying Dihexa to that axis were retracted in 2025 for falsified/fabricated figures, so the mechanism must be treated as unproven pending independent replication [4]

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