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Excessive ROS not only directly oxidatively damage mtDNA, leading to mutations and deletions, but also induce mitochondrial membrane lipid peroxidation and protein functional inactivation, triggering decreased mitochondrial membrane potential and cytochrome C release (Zhen et al., 2023

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10.1016/j.str.2023.05.019

Vogel DG, Malekzadeh MH, Cornford ME, Schneider JA, Shields WD, Vinters HV (1990) Central nervous system involvement in nephropathic cystinosis

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