In addition, we performed cell death reversal experiments using specific inhibitors targeting ferroptosis (Fer-1, Ferrostatin-1), oxidative stress (NAC, N-acetylcystein), necrosis (Nec-1, Necrostatin-1), apoptosis (ZVAD, Z-Val-Ala-Asp(O-Me)-fluoromethylketone), and autophagy (CQ, Baf-A1 and 3-MA) to identify the type of cell death induced by the combined treatment with KU-57788 and RSL3
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& Tchkonia, T
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Vitamin E synergizes with GPX4 in correcting the action of lipid hydroxyperoxides, and as a consequence may therefore limit perturbation of mitophagy in PKD
Through its free thiol groups, NAC can bind to mercury and form non-toxic, excretable complexes, thereby limiting tissue accumulation and associated oxidative damage
