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Ericson MC

Collectively, these metabolic alterations converge into a unified model of estrogen-deficiencydriven hepatic inflammation characterized by enhanced IDO1/TDO2KYN signaling, glycolytic reprogramming, phospholipid oxidation, TCA-cycle rewiring with itaconate accumulation, amino acid and purine pathway disruption, and redox imbalance through impaired PPP and taurine metabolism

[DOI] [PubMed] [Google Scholar] 23.Deanfield J., Verma S., Scirica B.M., Kahn S.E., Emerson S.S., Ryan D., Lingvay I., Colhoun H.M., Plutzky J., Kosiborod M.N., et al

These factors are crucial in the premature clearance of erythrocytes from circulation, a mechanism similar to that in HbE/-thalassemia [414]