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In dopaminergic cell models, resveratrol treatment increases IB levels (the inhibitory subunit that sequesters NF-B) and correspondingly lowers nuclear translocation of the NF-B p65 subunit (49)

For human succinic semialdehyde dehydrogenase, reversible disulfide bond formation of the active site cysteine (Cys 340 ) and an adjacent residue (Cys 342 ) was described as regulatory redox-switch mechanism

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