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Compared to the control group, the ethanol (ulcer) group revealed a significant decrease in gastric pErk-1 immunoexpression

10.1016/j.jns.2017.08.3235 284 LiuP.WuL.PengG.HanY.TangR.GeJ.et al

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In addition, our in vitro studies indicate a novel mechanism, that is GSTP can be phosphorylated at Ser184 and enter the nucleus under LPS stimulation, and GSTP interacted with HMGB1 to inhibit cPKC-mediated phosphorylation of HMGB1 and association between HMGB1 and CRM1 in the nucleus