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A CCK-8 assay demonstrated that HOXA2 overexpression suppressed the increase in HK-2 cell proliferation induced by TGF-1 (Fig

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It has been suggested that the anti-tTG2 antibodies found in the blood of individuals with CD might interact with tTG2 in the thyroid gland, potentially leading to the onset of thyroid disorders (52)

These findings have generated significant research interest in the compound as a tool for investigating HGF/c-Met-associated signaling networks, synaptic regulatory processes, and molecular pathways governing neuronal connectivity

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