This argues strongly in favor metabolic state-driven approach as a strategy to fight antibiotic-resistant bacteria and to increase and prolong the efficacy of antibiotic drugs
a patient gets an FMT or probiotic to establish a core healthy microbiome, then a targeted enzyme inhibitor to prevent that microbiome from producing a particular neurotoxin, plus a diet to feed it the right substrates
Gen Hosp Psychiatry
The niche where IGF-1 LR3 consistently outperforms: acute recovery from injury or training overreach
2 Redox biology and endocrine dysregulation in PCOS 2.1 OS pathways in PCOS: sources of ROS and antioxidant defense impairment Emerging evidence identifies PCOS as a disorder of redox dyshomeostasis, contributing to its complex reproductive, endocrine, and metabolic manifestations ( Among endogenous sources, mitochondrial electron transport chain (ETC) inefficiency stands out as a dominant ROS generator in PCOS ( Androgen excess emerges not only as a hormonal hallmark of PCOS but also as a direct ROS instigator ( While ROS sources increase, antioxidant defenses in PCOS are often found depleted ( 2.2 Mitochondrial dysfunction, lipid peroxidation, and redox imbalance in oocytes and granulosa cells The competence of oocytes and their surrounding granulosa cells is fundamentally governed by mitochondrial health, balanced redox homeostasis, and membrane integrity ( LPO is an evident indicator of oxidative injury in ovarian cells
After your first eight sessions, you can maintain the results with a monthly treatment
