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The identification of GLP-1R in other regions of the CNS provided a further advance in the understanding of the role played by GLP-1/serotonin cross-talk in the regulation of homeostatic control of body mass
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This fragmentation makes it nearly impossible to construct a cross-tissue causal model when each domain interprets the data through its own methodological and conceptual lens
10F) and the immune cell infiltrates were not observed (Fig
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