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Toxicol Lett 84:712 Corcoran GB, Wong BK (1986) Role of glutathionein prevention of acetaminophen-induced hepatotoxicity by N -acetyl-l-cysteine in vivo: studies with N -acetyl-d-cysteine in mice

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3B), as immune cells generate ROS as a primary defense mechanism against invading pathogens

The rationale is that the membrane fusion proteins on the infectious virus necessary to merge the viral with the host cell membrane are in prefusion conformation, exposing epitopes unique to this structure that can be targeted by neutralizing antibodies to prevent infection before the virus enters the cell

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