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Appropriate timing and amplitude of extracellular Ca 2+ influx and intracellular Ca 2+ release determines the rhythmicity of activation and the strength of contraction ( 2+ overload within the cardiomyocyte cytosol

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Co-culturing ESCs with monocyte-derived macrophages and NK cells from endometriosis patients showed that the interaction between ESCs and macrophages downregulates NK cell cytotoxicity by enhancing the release of IL-10 and TNF- (37), potentially allowing endometrial fragments to evade immune clearance (38)

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