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This increased tumor-targeting ability may be attributed to the significantly higher number of amino acid groups at the edges of LAAM GSH-CQDs, resulting in 3-4 times greater binding affinity with LAT1 compared to LAAM TC-CQDs (Supplementary Fig

These observations become important later when deciding whether to extend any escalation step

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Depletion of the novel p53-target gene carnitine palmitoyltransferase 1C delays tumor growth in the neurofibromatosis type I tumor model

Media reports echo this complexity