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Epidemiological studies reported an almost two-fold increased risk for infected individuals to develop CRC, but only recently, direct causal and functional links between the chronic infection and CRC have been revealed

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In particular, they produce ATP, which leads to falsely high results when AK inhibitors in the CK reaction are exhausted

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Furthermore, subchondral angiogenesis, often driven by VEGF and mechanical stress, disrupts the osteochondral interface and promotes cartilage deterioration (Noh et al., 2020)

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