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The capsule polysaccharide from serogroups A, B, C, W, and Y meningococci inhibits C1q interaction with bound anti-fHbp and anti-porin A antibodies, and, consequently, the deposition of C4b and classical complement pathway (Jarvis and Vedros, 1987), as well as LOS sialylation (Vogel et al., 1997), limits C3 deposition, inhibiting the alternative pathway

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Our group has previously reported that the type of pathogen present within ACs can induce the production of different soluble mediators by BMDCs during efferocytosis

Talib, W

They wrote: Remarkably, under the optimized micelle conditions, A42 assembles into oligomers that insert into lipid bilayers as well-defined pores and adopt a specific structure with characteristics of a -barrel arrangement (Zhaliazka et al., 2023)

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