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In AD, mitochondrial abnormalities, including impaired electron transport chain function and reduced mitochondrial biogenesis, lead to increased ROS production and diminished ATP generation

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This process, known as Phase I and Phase II detoxification, creates a significant amount of "oxidative stress." Think of this stress like the exhaust coming from a car engine

For testosterone, data imply support for Leydig cell function and steroid output under stress conditions

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