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CD36 was enriched in dysfunctional TILs, and FA uptake by CD36 results in reduced secretion of effector molecules such as IFN-, GZMB, and TNF in mice with melanoma colon adenocarcinoma [51] and patients with non-small cell lung cancer [48]

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While the adaptive unfolded protein response (UPR) initially aims to restore proteostasis, persistent or excessive ER stress in PD switches the cellular outcome to PCD, promoting dopaminergic neuron apoptosis (11, 12)

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