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Although the predominance of available literature from in vivo ischemic studies has focused on the involvement of synaptic vesicle zinc, it is likely that intracellular zinc accumulation during ischemia is achieved through a possible synergism of zinc release from both synaptic and intracellular stores of zinc, as most recently demonstrated during global ischemia (38) and reported in other experimental paradigms (4551)

This mechanism explains why PT-141 works differently than anything else

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Mechanistically, the neuroprotective effect is mediated by the PI-3K activation (Hossain et al., 2002)

Metoprolol belongs to a class of drugs known as beta-blockers, specifically cardioselective beta-1 adrenergic receptor blockers

Reversion to injections is easy if needed