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In mouse models of type 1 diabetes, verapamil promoted survival of functional beta cells via a mechanism that involves reduced expression of the cellular redox regulator thioredoxin-interacting protein [102]

Mitzner explains that this hormone is released after eating and regulates blood sugar, slows digestion and signals fullness to the brain. As a result, people taking these medications typically experience reduced hunger and therefore eat fewer calories

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doi: 10.1016/s0891-5849(02)00826-2

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