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Regarding fibrosis, while direct anti-fibrotic mechanisms are less established, preclinical studies indicate that GLP-1 RAs could suppress fibrogenic cell activation and reduce expression of pro-fibrotic genes encoding extracellular matrix proteins, potentially facilitating fibrosis regression in conjunction with improvements in steatosis and inflammation [29-32]

0.820.96), whereas SGLT2is showed a significant reduction in HF hospitalization (0.72

Diabetes Care, 46(Supplement_1), S1S322

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Liraglutide for managing overweight and obesity Recommendations chapter (TA664)