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Mechanism of ferroptosis in traditional Chinese medicine for clinical treatment: a review

This pro-migratory and proliferative effect is closely associated with the activation of the FAK-paxillin pathway

doi:10.1093/aje/kww212

Compared to classical gastroprotective peptides such as sucralfate or proton pump inhibitors (PPIs), it acts via a completely different pathway: restoration of mucosal microcirculation rather than acid reduction or formation of a mechanical protective layer

Gclc f/f mice (hereafter described as Gclc WT) were also treated with tamoxifen to control for potential confounding effects associated with tamoxifen treatment

This produces harmful reactive oxygen species (ROS) or free radicals and depletes glutathione