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Our results indicate that VRK2 phosphorylates COPII component Sec24C and promotes its interaction with SLC7A11, thus facilitating ER-to-Golgi transport and membrane expression of SLC7A11

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Unlike traditional medications such as disulfiram, naltrexone, and buprenorphine, which primarily target neurotransmitter pathways directly associated with addiction, GLP-1RAs modulate reward mechanisms indirectly through metabolic and neuroendocrine pathways

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