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Both SP and L-car attenuated DOX-induced ovarian oxidative damage, subsequent ovarian mtDNA expression, and activation of downstream inflammatory and apoptotic pathways, particularly lowering P53 and caspase-3 protein expressions in preantral follicles, thus preventing ovarian insult and follicle demise and extending the post-chemotherapy fertile period
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High epidemiological reliability denotes the strongest available evidence with no indication of significant variance or bias ( (i) Persuasive: statistical significance per the random-effects model of p 1,000 cases, low heterogeneity among the selected studies ( I 2 1,000 cases, and most studies indicating a significant effect
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