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22 ChenCYanYWuYLuMXingYBaiYet al
Exp Op Drug Disc
Additionally, if OCSCs are exposed to inflammatory mediators, undergo mesenchymal endothelial transformation, or experience dysregulation of the redox balance in the tumor microenvironment (TME), they may stimulate the continuous proliferation of OCSCs, potentially leading to OC development (63)
Notably, E and progesterone, as key regulators of uterine fibroids, have been demonstrated to activate the PI3 K/Akt pathway and promote the proliferation of fibroid cells [83]
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