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Conclusion: Overall, this study sheds light on metabolic pathways that are dysregulated in tamoxifen-resistant cell lines and the potential role of each of these pathways in the development of resistance

This modified GHRH(1-29) peptide has been engineered with amino acid substitutions to increase its biological half-life and resistance to enzymatic degradation by dipeptidyl peptidase-IV (DPP-IV)