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Stress increases energy demands in the brain, and though vital, energy production can be a messy job, creating a lot of free radicals.[1][2] Add to this inflammation from numerous sources, and something needs to be present to counterbalance it

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This mechanism is particularly significant for lupus pathogenesis, given that DN T cells are a primary source of pro-inflammatory IL-4, IL-17 and necrotic debris (Lai et al