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In 1906 , Dr

4 Division of Pulmonary Medicine and Allergy and Immunology, Medical University of South Carolina, Charleston, SC, USA

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First, under normal conditions, elevated HO1 expression attenuates oxidative stress to exert cytoprotective effects, and second, sustained HO1 upregulation releases ferrous iron to exacerbate oxidative stress, which leads to ROS accumulation through the Fenton reaction