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J Inorg Biochem

Targeted molecular pathways involved include thyroid follicular cells, TSH receptors, and thyroid hormone receptors

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The resulting oxidative overload disrupts redox-sensitive pathways (e.g., NF-B, Nrf2), promoting inflammation, fibrosis, and cellular apoptosis ( Elevated levels of oxidative biomarkerssuch as malondialdehyde (MDA), 8-hydroxy-2-deoxyguanosine (8-OHdG), and F2-isoprostanesreflect systemic OS and correlate with HTN severity and DM complications (Spoto et al., 2025)

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