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The second limitation is pharmacokinetic in nature

219 It has been postulated that the ability of GLP-1 to enhance metabolism may decrease endoplasmic reticulum stress via increasing mitochondrial production of ATP and Ca 2+ , which could be utilised for assisting endoplasmic reticulum homeostasis

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Phosphorylation of T 495 residue on eNOS, which has traditionally been shown as a regulatory site inhibiting eNOS-derived nitric oxide production while promoting eNOS uncoupling and superoxide formation, was increased in animals who received PAGln (Supplementary Fig