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Additionally, several factors commonly present in humans and animals with CKD can exacerbate OS, including the activation of the reninangiotensin system, systemic hypertension, chronic inflammation, proteinuria, anemia, and advanced age [187,190]

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Expression of A53T mutated -synuclein in astrocytes leads to BBB disruption in mice and a decrease in glutamate transport in affected astrocytes [269]

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FIGURE 3 Together, these findings suggest that HA + GSH-C4 induces a transient, reversible cytokine activation with repair-associated Th1/Th17 features, with IFN- and IL-12p70 persisting even after crossover to HA alone

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