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Importantly, no signs of toxicity or adverse histologic changes were observed, indicating that thickening was part of a healthy adaptive response

Thiamin deficiency has been linked to increased -amyloid production in cultured neuronal cells and to plaque formation in animal models (65, 66)

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Traumatic brain injury and cerebral ischemia In a mouse traumatic-brain-injury model, BPC-157 including when given prophylactically before injury was associated with less intense traumatic lesions (subarachnoid and intraventricular hemorrhage, laceration), reduced consecutive brain edema, and an improved conscious/unconscious/death distribution

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