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Phosphorylation of Ser137 in the S3-node by CDK5 primes CaMKK2 for subsequent phosphorylation on Ser133 and Ser129 by GSK3, which results in inhibition of CaMKK2 activity [40]

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[19] All adverse events including musculoskeletal complaints must be recorded and assessed for potential relatedness to the investigational product by the trial team