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Reducing mechanical load on compromised joints while using BPC-157 to support tissue repair is a more complete approach than either intervention alone

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It modulates iron levels by inducing ferritin expression, thereby limiting iron-catalysed free radical generation

Separate studies have reported that BPC-157 modulates the nitric oxide (NO) system across multiple injury models, opposing the deleterious effects of NO synthase inhibition by L-NAME and reinforcing the effects of NO substrate by L-arginine

Best results occur with specific types of bone defects and when combined with excellent post-operative care