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Studies on animal models have shown that Ipamorelin stimulates the secretion of growth hormone (GH) from somatotropic pituitary cells in a selective manner without the accompanying significant increase in cortisol, aldosterone or prolactin concentrations, which is observed with the administration of other GHRPs, such as GHRP-6 or GHRP-2

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AC system activation generates an increase of the intracellular secondary messenger, cyclic adenosine monophosphate (cAMP), through the conversion of adenosine triphosphate, which then results in the activation of protein kinase A (PKA) and exchange protein activated by cAMP (Epac) [32,33,34,35]

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