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Structural basis for GPR40 allosteric agonism and incretin stimulation

Individuals with NAFLD: Those with non-alcoholic fatty liver disease, especially when linked to obesity, due to the medications potential to reduce liver fat

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A Phase I safety trial evaluating the administration of synthetic T4 at massive doses ranging from 42mg to 1260mg over 14 days demonstrated excellent tolerability with no severe toxicities or serious adverse events reported in healthy adults

The drug works primarily by mimicking the incretin hormone GLP-1, which enhances glucose-dependent insulin secretion, suppresses glucagon release, slows gastric emptying, and reduces appetite through central nervous system pathways