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In addition to the inhibition of cellular senescence, which was the first observation providing mechanistic insights into the role of KDM2B in oncogenesis [2,4], our studies addressing the effects of the KDM2B knockdown in a set of ten cancer cell lines, showed that KDM2B promotes cell cycle progression in all and inhibits apoptosis in some [10]

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Additionally, studies have further demonstrated that an increased SDF-1 level in ischemic area played a crucial role for EPC homing to the ischemic area for angiogenesis and neovascularization [35, 36]

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