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In accordance with its reduced capacity for stimulating insulin release, exendin-asp3 failed to reduce apoptosis following glucolipotoxicity 24 or endoplasmic reticulum stress 25 , both involved in T2D pathogenesis

Sigma-Aldrich, Madrid, Spain) and cultured in RPMI 1640 medium supplemented with 10% heat-inactivated FBS, penicillin, streptomycin, L-glutamine, and 50 U/mL of human recombinant interleukin 2 (IL2) [49]

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