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A3: Research typically characterizes IGF-1 and its analogues, including IGF-1 LR3, as supporting hyperplasia (increased cell number) rather than direct hypertrophy (increased cell size)

and the fact that the more traditional choice for nasal sprays of microparticles cannot penetrate the mucin (3941)

doi:10.1007/s10815-012-9714-1

Soudja, S

A Multi-Pathway Model We can think of this as a convergence of several mechanisms: L-cell activation from viral or microbial sensing Vagal signaling altered by cytokine release Gut-brain axis dysregulation, mirroring GLP-1 agonist effects Delayed motility from enteric nervous system inflammation Together, these pathways could produce a temporary GLP-1-like state during and shortly after the viral illness