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This is, then, oxidized by cytosolic isocitrate dehydrogenase 1 (IDH1) to -ketoglutarate with the production of NADPH required for the detoxification from reactive oxygen species (ROS

These molecules are primarily formed in mitochondria during respiration, but additional sources include redox metal ions and enzymatic reactions [24]

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However, the clinical difference in systemic delivery is significant

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By activating telomerase, epithalon can reduce the shortening of telomeres, theoretically helping you age backward. I personally use the Khavinson Protocol for dosing epithalon (yes, the very same aforementioned professor), which uses the exact dosage from a fifteen-year longevity study in humans that produced impressive results for controlling telomere shortening