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Invariant natural killer T cells contribute to chronic-plus-binge ethanol-mediated liver injury by promoting hepatic neutrophil infiltration

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doi: 10.1016/j.ecoenv.2019.03.036

Their catabolism may in turn release a myriad of uremic toxins (e.g., 4-hydroxyphenyl-acetic, -pyruvic, and -lactic acids) and pro-inflammatory compounds (e.g., kynurenic acid), which modulate oxidative stress and inflammation [39]

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