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This triggers: Dopamine release in reward/motivation circuits Enhanced neural signaling for sexual desire Increased genital arousal through central (not peripheral) mechanisms This is fundamentally different from PDE5 inhibitors (Viagra, Cialis) which work on blood vessels

Based on these findings the authors concluded 'that central GLP-1 is a new physiological mediator of satiety' (Turton, O'Shea, Gunn)

Mehan, S

It relies on specific nutrients to remain active

Consequently, IGF-1 LR3 has improved potency and metabolic stability [5]