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mtDNA and ROS released during ferroptosis activate the NLRP3 inflammasome, triggering caspase-1 activation and GSDMD cleavage

When administered at therapeutic levels, APAP is primarily metabolized via the glucuronidation pathway, producing non-toxic metabolites that the liver and kidneys can further process (Chiew and Isbister 2023)

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aureus using in vitro assays ( Clostridium botulinum type A (Nidadavolu et al., 2012)

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Sodium valproate is associated with increasing ammonia levels and decreasing carnitine status, and carnitine intervention has been found to normalise both