Aug 2022;31(4):e13628
Nicotinamide mononucleotide adenylyl transferase-mediated axonal protection requires enzymatic activity but not increased levels of neuronal nicotinamide adenine dinucleotide
However, MT substantially inhibits this early cardiac apoptosis caused by diabetes by suppressing mitochondrial oxidative stress, particularly the depletion of GSH, which significantly prevents the development of DCM
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