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As individuals age, the capacity for glutathione synthesis naturally declines, creating a progressive deficit that may contribute to age-related cellular dysfunction and disease susceptibility

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This results in lower expression of cytokines (TNF-, IL-6, etc.) and adhesion molecules, dampening the vascular inflammation that drives atherosclerosis and hypertensive organ damage (Wang Q

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Glutathione plays a critical role in protecting mitochondria from oxidative stress, supporting metabolic functions, and regulating apoptosis

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